Eye Diseases & Conditions

Severe protozoal infection, often contact lens-related with ring infiltrate.

Leading cause of central vision loss in older adults — dry and wet forms.

Most common eyelid malignancy, locally invasive with pearly borders.

Chronic inflammation of the eyelid margins, often staphylococcal or meibomian-related.

Sudden sectoral vision loss from arterial embolus or thrombosis.

Venous blockage causing macular edema and hemorrhage.

Endothelial decompensation causing persistent corneal edema and bullae.

Acute painless profound vision loss — cherry-red spot sign.

Severe retinal hemorrhage and ischemia, high risk of neovascularization.

Serous detachment of macula, often stress-related self-resolving.

Congenital retinal telangiectasia with exudation, mainly unilateral in boys.

Epithelial defect from trauma, causing acute pain and foreign body sensation.

Inherited bilateral opacities (e.g. Fuchs, lattice, granular).

New vessel ingrowth, often from hypoxia or inflammation.

Vascularized fibrosis at limbus, common in chronic blepharitis or contact lens overuse.

Infectious or sterile stromal loss, urgent sight-threatening condition.

Fluid cysts in macula post-surgery, uveitis, or vascular disease.

Excess upper eyelid skin, often age-related, impairing superior field.

Microvascular complications — non-proliferative to proliferative stages.

Tear film dysfunction causing ocular surface damage and discomfort.

Outward turning of eyelid, leading to exposure keratopathy.

Inward turning of eyelid, causing trichiasis and corneal damage.

Congenital inward rotation of eyelid margin, common in Asian children.

Fibrocellular membrane causing macular pucker and distortion.

Recurrent dendritic epithelial or stromal disease from HSV.

VZV reactivation affecting cornea, often with skin vesicles.

Acute painful eyelid lump — stye (external) or internal chalazion precursor.

Retinal arteriolar narrowing, AV nicking, hemorrhages from high BP.

General corneal inflammation — microbial, autoimmune, or exposure-related.

Progressive corneal thinning and ectasia, leading to irregular astigmatism.

Congenital synkinesis — eyelid elevation with jaw movement.

Benign squamous epithelial tumor, often viral (HPV).

Peripheral inferior corneal thinning with high against-the-rule astigmatism.

Drooping upper eyelid due to levator dysfunction or neurogenic causes.

Neurosensory separation — rhegmatogenous, tractional, or exudative.

Full-thickness break, high risk for progression to detachment.

Inherited rod-cone dystrophy with night blindness and tunnel vision.

Most common intraocular malignancy in children, often RB1 mutation.

Abnormal retinal vascularization in preterm infants.

Chronic facial/ocular inflammation with telangiectasia and meibomian dysfunction.

Rare, aggressive malignancy mimicking chronic blepharitis.

Peripheral ischemia and sea-fan neovascularization in sickle hemoglobinopathies.

Aggressive skin cancer of the eyelid, may metastasize.

Diffuse epithelial erosions, often from dry eye or toxic exposure.

Misdirected eyelashes rubbing against the cornea, causing irritation and abrasion.

Yellowish cholesterol deposits on eyelids, marker for hyperlipidemia.